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The Slimming Science

AimSlim™ may reduce appetite and stored fat levels by the following dual mechanisms of action:

Satiety effect by Vagus Nerve stimulation:

Achyranthes Aspera seeds have protein, fatty acids like Oleic Acid and Linoleic Acid, and Saponins. These compounds, especially oleic acid (once converted to Oleoylethanolamide – OEA) can help stimulate the vagus nerve once inside the GI tract to promote a feeling of satiety (the “I’m full feeling”).

Fat Reduction Action by Stimulation of Lipolysis:

It is also believed that OEA among other compounds can stimulate lipolysis (fat metabolism). Fat metabolism can provide sustained energy, which could be much more helpful in battling any appetite induced fatigue.

It is important to note that we believe the combined effects of actives and co-factors and co-nutrients is what leads to results.

 
There are numerous published research studies that attempt to show the mechanism of saponins, Oleic Acid (converted to OEA), and Lenoleic Acid stimulating the vagus nerve and causing appetite suppression, perhaps the most famous being:
  • Central Nervous System Agents in Medicinal Chemistry: Brain Molecules and Appetite: The Case of Oleoylethanolamide
  • Nature: Oleylethanolamide [OEA] regulates feeding and body weight through activation of the nuclear receptor PPAR-α
  • Science Direct: The Lipid Messenger OEA Links Dietary Fat Intake to Satiety
There are also published research studies that show OEA and saponins being effective for fat metabolism:
  • The Journal of Biological Chemistry: Oleoylethanolamide Stimulates Lipolysis by Activating the Nuclear Receptor Peroxisome Proliferator-activated Receptor α (PPAR-α)
  • Science Direct – Neuropharmacology: Oleoylethanolamide, an endogenous PPAR-α agonist, lowers body weight and hyperlipidemia in obese rats

These statements have not been evaluated by the Food and Drug Administration.
This product is not intended to diagnose, treat, cure, or prevent any disease.